stercoralis. Treatment involving albendazol of 400 mg/day and steroid of 0.5 was initiated. In the controls performed at the first month of treatment a clinic improvement was seen but radiology was stable. Bronchoscopy was repeated and transbronchial biopsy showed that the structures within the granuloma were thickened and broken. ( Fig. 4) This case is reported since it is rarely seen and hardly diagnosed. Communications between PF01367338 the pathologist and parasitologist allowed the diagnosis. Although S. stercoralis can also be seen in temperate climates, it is present mainly in tropical and subtropical climates. Strongyloidiasis affects anywhere from 30 to 100 million people

worldwide [3] and [4] and is endemic in Southeast Asia, Latin America, sub-Saharan Africa, and parts of the southeastern United States [3] and [5]. In Turkey, it is reported as sporadic cases, particularly in immunosuppressed individuals. It is seen in regions where the soil is humic, at temperatures

above 20 °C and with long-term high humidity. Individuals who work or wander barefoot in adobe, brick or tile manufacturing sites, mines, irrigated farming areas, streams and marsh waterfronts are most commonly infected [1]. The onset of infection is mostly with access of filariform larvae through the skin. It may also result from autoinfection and larvae access through the digestive tract [6] and [7]. The patient presented herein had barefoot soil contact in a holiday resort as well as history of pica and habit of BGB324 eating clams. The infection might have occurred through the skin or digestive system. Larvae accessing the host through the skin travel directly to the lungs via blood vessels. After spilling into the alveolar space, the larvae advance through the trachea and pharynx, where they are swallowed. They are later adsorbed on the duodenal and upper jejunal mucosa. They complete their maturation by approximately two weeks and larvae-containing ovum start to grow within

mature females. Rhabditiform larvae emerging from the ovum shortly after pass into the intestinal lumen and are discharged via the excreta [1]. Given the migration pathway followed within the host, the primary signs and symptoms involve the skin, Liothyronine Sodium lungs and the gastrointestinal system. Clinical findings vary depending on the amount of parasites exposed, the immune status of the host and the body part involved. Disseminated disease or hyperinfection may develop in immunosuppressed individuals, in whom mortality rates can be as high as 87% [8]. Individuals with normal immune system may be asymptomatic or may present as acute or chronic vases with pulmonary or gastrointestinal symptoms [1]. Presence of pulmonary symptoms in our patient may be due to the intact immune system and/or non-chronic exposure to the infection.